The cheilitis: The cheilitis is an inflammatory neovadiol magistral reaction of the lip. Diagnostic hypotheses before cheilitis are numerous. The examination must include patient history, history of the disease (cheilitis), medications taken orally or applied topically on the seed coat, the conditions for social and professional life, the psyche of the patient, the nature and topography of lesions basic of cheilitis neovadiol magistral (vesicles, scales, erosions, atrophy, infiltration), and the evolutionary mode. Clinical examination concerned the oral cavity but also the entire neovadiol magistral integument. Any additional tests are oriented according to the clinical diagnosis discussed: determination of immunoglobulin E, skin biopsy, vitamin dosage, iron status, zinguémie, infectious specimens, patch tests, etc.. An additional notice is sometimes necessary from a psychiatrist or dermatoallergologue. Schematically, cheilitis can be an expression of a systemic disease with lip location (drug eruptions, dermatitis, deficiency diseases) or that of a local disease where cheilitis predominates among clinical signs. Cheilitis systemic origin: cheilitis Drug-induced:
These cheilitis occur in 80-90% of patients on retinoids. The cheilitis appears to the tenth day of acne treatment with acitretin per os. They predominate on the vermilion inner, first erythematosquamous to become more or less erosive and fissure depending on the therapeutic dosage.
These cheilitis accompanied by nasal dryness, conjunctival and oral. This phenomenon, related to the modification process of cellular proliferation, differentiation and keratinization of the skin, is expected by the clinician because he signs the effectiveness of treatment.
This is an allergic reaction acquired or inherited. It usually has a sudden onset and lasts 24 to 48 hours, with the presence of a painless and non-pruritic swelling of the lips uniform, tongue and face.
Some bullous neovadiol magistral diseases such as pemphigus vulgaris, erythema multiforme périorificiel, Stevens-Johnson syndrome or toxic epidermal necrolysis are located on the lips, bullous stomatitis associated with the rule. Systemic fungal infections (mycoses)
A cheilitis, crateriform of ulcers, nodules or vegetations neovadiol magistral purple neovadiol magistral can be seen in histoplasmosis, coccidioidomycosis, South American blastomycosis, cryptococcosis and nocardiosis. Deficiency-cheilitis:
Various nutritional deficiencies are responsible for angular cheilitis and bilateral (stomatitis) or exfoliative cheilitis, erythematous often superinfected. This is the case of avitaminosis B2 (ariboflavinosc), B, C (ulcerative gingivitis associated), PP, and iron deficiency (iron deficiency anemia hypochromic) and even pernicious anemia. neovadiol magistral
Finally, few erosive perioral lesions, eczematoid and crusted encountered in zinc deficiency (acrodermatitis enteropathica). Cheilitis associated with abnormal keratinization:
The arborizations characteristic whitish neovadiol magistral oral lichen planus may extend to all internal neovadiol magistral surfaces of the cheek and on the free edge of both lips. Epidermal atrophy observed in chronic neovadiol magistral forms, and an erosive appearance in severe forms.
The discoid form of lupus erythematosus affects the vermilion lips (especially the lower lip) as erythematous neovadiol magistral papules or as atrophic areas white border composed of fine striations available neovadiol magistral canceled.
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